Here are a few of the tests:

The most common congenital cardiovascular defects can be grouped into several anatomic categories including:

  • Tricuspid Valve Dysplasia

  • Malformation of the atrioventricular valves

  • Malformation of the ventricular outflow leading to obstruction of blood flow

  • Defects of the cardiac septa (shunts)

  • Abnormal develop of the great vessels or other vascular structures

  • Complex, multiple, or other congenital disorders of the heart, pericardium, or blood vessels

Please visit the

Orthopedic Foundation for Animals!

Hip Dysplasia typically develops because of an abnormally developed hip joint, but can also be caused by cartilage damage from a traumatic fracture. OFA classifies hips into seven different categories: Excellent, Good, Fair (all within Normal limits), Borderline, and then Mild, Moderate, or Severe (the last three considered Dysplastic).

  • Excellent: Superior conformation; there is a deep-seated ball (femoral head) which fits tightly into a well-formed socketacetabulum) with minimal joint space.

  • Good: Slightly less than superior but a well-formed congruent hip joint is visualized. The ball fits well into the socket and good coverage is present.

  • Fair: Minor irregularities; the hip joint is wider than a good hip. The ball slips slightly out of the socket. The socket may also appear slightly shallow.

  • Borderline: Not clear. Usually more incongruency present than what occurs in a fair but there are no arthritic changes present that definitively diagnose the hip joint being dysplastic.

  • Mild: Significant subluxationpresent where the ball is partially out of the socket causing an increased joint space. The socket is usually shallow only partially covering the ball.

  • Moderate: The ball is barely seated into a shallow socket. There are secondary arthritic bone changes usually along the femoral neck and head (remodeling), acetabular rim changes (osteophytes or bone spurs) and various degrees of trabecular bone pattern changes
    (sclerosis).

  • Severe: Marked evidence that hip dysplasia exists. Ball is partly or completely out of a shallow socket. Significant arthritic bone changes along the femoral neck and head and acetabular rim changes

No one can predict when or even if a dysplastic dog will start showing clinical signs of lameness due to pain.

A general term used to identify an inherited polygenic disease in the elbow. Three specific etiologies make up this disease and they can occur independently or in conjunction with one another. These etiologies include:

  • Pathology involving the medial coronoid of the ulna (FCP)

  • Osteochondritis of the medial humeral condyle in the elbow joint (OCD)

  • Ununited anconeal process (UAP)​

​Visit OFA for information on elbow grades, there are no grades for a radiographically normal elbow. The only grades involved are for abnormal elbows with radiographic changes associated with secondary degenerative joint disease.

Exercise-Induced Collapse (EIC) is a genetic neuromuscular disorder characterized by muscle weakness, lack of coordination and life-threatening collapse after intense exercise in otherwise apparent healthy dogs.

A small proportion of labradors have an allele at the D of dilute locus that causes black Labradors to be blue or gray and brown dogs to be a pale grayish brown. Such dilute dogs have two d alleles at this locus. Dogs with a Dd genetic makeup do not have a detectable change in their coat color and therefore DNA testing is necessary to detect these carriers or heterozygotes of the dilute allele. Dilute colored dogs are not shown in many countries/associations. Danrich Labradors is NO TO DILUTES!

(CNM), previously known as HMLR, or Hereditary Myopathy, is an autosomal recessive mutation that causes insufficient muscle function in the Labrador Retriever breed. ... A dog can also be a carrier (CNM/n) of this disease, and will not display any symptoms.

Progressive retinal Atrophy, progressive Rod-cone degeneration (PRA-prcd) is a late onset, inherited eye disease affecting many breeds of dog. PRA-prcd occurs as a result of degeneration of both rod and cone type Photoreceptor Cells of the Retina, which are important for vision in dim and bright light, respectively.

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